Term
| Most important aspect of Ca |
|
Definition
|
|
Term
| Important because you can form insoluble PO4 precipitates which can clog blood vessels |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| Neuromuscular irritability |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Paresthesias (pins and needles) |
|
Definition
|
|
Term
| Tetanic muscle contractions (hands feet, larynx) |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| Decreased neuromuscular excitability |
|
Definition
|
|
Term
| Decreased neurotransmission |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
| Hypercalcemia or Hypocalcemia |
|
|
Term
| Hypercalciuria (kidney stone) |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Increased with low free Ca |
|
Definition
|
|
Term
| Overall effect is to increase plasma Ca and decrease plasma PO4 |
|
Definition
|
|
Term
| Decreased with high free Ca |
|
Definition
|
|
Term
| Most immediate effect of PTH |
|
Definition
|
|
Term
| Effect of PTH requiring changes in osteoblasts and osteoclasts |
|
Definition
| Bone reabsorption (Ca, PO4) |
|
|
Term
| This organ effected by PTH reabsorbs Ca and secretes PO4 |
|
Definition
|
|
Term
| Inhibits MP differentiation into osteoclasts |
|
Definition
|
|
Term
| Tumor causing hypercalcemia |
|
Definition
|
|
Term
| Kidney stones, broken bones, abdominal groans, psychic moans |
|
Definition
|
|
Term
| Most commonly Iatrogenic (surgery), Autoimmune causing hypocalcemia |
|
Definition
|
|
Term
| Increases release of calcitriol |
|
Definition
|
|
Term
| Most important aspect is to regulate PO4 levels |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| Increases Ca, PO4, Mg absorption from GI |
|
Definition
|
|
Term
| Improves mineralization, regulating organic matrix by inhibiting collagen |
|
Definition
|
|
Term
| If absent, collagen deposits=soft bendy bones |
|
Definition
|
|
Term
| 1st symptom of hypercalcemia |
|
Definition
|
|
Term
| Stimulated in response to hypercalcemia to reduce Ca and PO4 levels |
|
Definition
|
|
Term
| Main target is inhibiting activity of osteoclasts |
|
Definition
|
|
Term
| Main source is from diet or GI tract |
|
Definition
|
|
Term
| gi>glucose>increase ATP>decrease K>depolarize>increase Ca influx>increase _____ |
|
Definition
|
|
Term
| Second messenger increasing insulin release |
|
Definition
|
|
Term
| Released by beta cells in the pancreas |
|
Definition
|
|
Term
| Stimulates insulin release by decreasing K |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Secretin effect on insulin |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| Glucagon effect on insulin |
|
Definition
|
|
Term
| Glucose effect on insulin |
|
Definition
|
|
Term
| Somatostatin effect on insulin |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Exercise effect on insulin |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Insulin effect on ketogenesis |
|
Definition
|
|
Term
| Insulin effect on glycogenesis |
|
Definition
|
|
Term
| Insulin effect on protein synthesis |
|
Definition
|
|
Term
| Insulin effect on lipogenesis |
|
Definition
|
|
Term
| Insulin effect on glycolysis |
|
Definition
|
|
Term
| Insulin effect on glycogenolysis |
|
Definition
|
|
Term
| Insulin effect on protein degradation |
|
Definition
|
|
Term
| Insulin effect on lipolysis |
|
Definition
|
|
Term
| Insulin effect on gluconeogenesis |
|
Definition
|
|
Term
| Insulin effect on ketogenesis |
|
Definition
|
|
Term
| Glucagon effect on glycogenesis |
|
Definition
|
|
Term
| Glucagon effect on protein synthesis |
|
Definition
|
|
Term
| Glucagon effect on lipogenesis |
|
Definition
|
|
Term
| Glucagon effect on glycolysis |
|
Definition
|
|
Term
| Glucagon effect on glycogenolysis |
|
Definition
|
|
Term
| Glucagon effect on protein degradation |
|
Definition
|
|
Term
| Glucagon effect on gluconeogenesis |
|
Definition
|
|
Term
| Glucagon effect on ketogenesis |
|
Definition
|
|
Term
| Cortisol effect on glycogen syntase |
|
Definition
|
|
Term
| If you have low glucose between meals, CNS response is to increase ___ and alpha cells will release ____ |
|
Definition
|
|
Term
| Insulin effect on glucagon |
|
Definition
|
|
Term
| Insulin effect on somatostatin |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Autoimmune disease that is insulin dependent |
|
Definition
|
|
Term
| Not insulin dependent, but involves a reduced sensitivity to insulin |
|
Definition
|
|
Term
| Immunodeficiency involving ketosis but not obesity |
|
Definition
|
|
Term
| Disease involving obesity but rarely ketosis |
|
Definition
|
|
Term
| Age is a good indicator of type 1 vs type 2 diabetes mellitus |
|
Definition
|
|
Term
| Good measurement of Insulin because it has a long half life and is not present in the injected form |
|
Definition
|
|
Term
| Insulin uses this type of receptor |
|
Definition
|
|
Term
| CTH, glucagon, PTH use this type of receptor |
|
Definition
|
|
Term
| GH and prolactin use this type of receptor |
|
Definition
|
|
Term
| cAMP uses this type of receptor |
|
Definition
|
|
Term
| cGMP uses this type of receptor |
|
Definition
|
|
Term
| T3/T4 use this type of receptor |
|
Definition
| Cytosolic, steroid transcription |
|
|
Term
| E uses this type of system |
|
Definition
|
|
Term
| With a blood glucose of 129, glucagon (normal 40-60)levels should be |
|
Definition
|
|
Term
| With a blood glucose of 60, glucagon (normal 40-60) should be |
|
Definition
|
|
Term
| What would cause: low blood glucose, low-normal glucagon, high insulin |
|
Definition
| Insulin secreting tumor (beta cell) |
|
|
Term
| How do you differentiate an insulin overdoes from a beta cell tumor |
|
Definition
|
|
Term
|
Definition
|
|
Term
| When are GH levels the highest |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Dec glucose>SNS>NE, E>___ |
|
Definition
|
|
Term
|
Definition
|
|
Term
| GH acts on ___, which acts on ___, which increases ____, which stimulates growth |
|
Definition
| Janis kinase, Stat-stat, IGF1 |
|
|
Term
| Negative feedback of GHRH |
|
Definition
|
|
Term
|
Definition
|
|
Term
| How do you treat laron dwarf |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Glucagon is released from ___ cells of the pancreas |
|
Definition
|
|
Term
| Cortisol effect on glycogen synthase |
|
Definition
|
|
Term
| Cortisol effect on inflammation |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Hypoglycemia effect on cortisol |
|
Definition
|
|
Term
| What time of day are cortisol levels the highest |
|
Definition
|
|
Term
| Cytokine effect on cortisol |
|
Definition
|
|
Term
| Stress effect on cortisol |
|
Definition
|
|
Term
| If you have a lack of cortisol you can go into |
|
Definition
|
|
Term
| What would cause low CRH, high ACTH, high Cortisol |
|
Definition
|
|
Term
| What would cause low CRH, low ACTH, low or undetectable Cortisol |
|
Definition
|
|
Term
| What would cause low CRH, low ACTH, high Cortisol |
|
Definition
|
|
Term
| Stimulates bone reabsorption, inhibits bone formation and Ca reabsorption from the kidney |
|
Definition
|
|
Term
| Patients taking this drug should supplement Ca and Vit D |
|
Definition
|
|
Term
| Disease associated with low Ca levels and low PTH levels |
|
Definition
|
|
Term
| Disease associated with high Ca levels and low PTH levels |
|
Definition
| Secondary non-PTH hypercalcemia |
|
|
Term
| Disease associated with low Ca levels and high PTH |
|
Definition
|
|
Term
| Disease associated with high Ca levels and high PTH levels |
|
Definition
|
|
Term
| In children RIckets is due to a deficiency in what |
|
Definition
|
|
Term
| Vit D excess leading to hypercalcemia leads to what |
|
Definition
|
|
Term
| Released in response to gastrin after eating to prevent excess Ca and PO4 levels |
|
Definition
|
|
Term
| Produced by parafollicular cells of the thyroid gland |
|
Definition
|
|
Term
| Where are the parathyroid glands located |
|
Definition
| Posterior surface of thyroid |
|
|
Term
| Ms. C has a total plasma Ca of 2.1 mM/L (normal 2.2-2.6 mM/L), at which blood pH will she be most likely to exhibit the most severe signs of hypocalcemia |
|
Definition
|
|
Term
| Ms. C has a total plasma Ca of 2.1 mM/L (normal 2.2-2.6 mM/L). What would you expect of her PTH blood levels |
|
Definition
|
|
Term
| What is the effect of PTH on the kidney |
|
Definition
| Increased Ca reabsorption |
|
|
Term
| E acts on betaAR to increase heart rate by |
|
Definition
|
|
Term
| E acts on alpha2AR to decrease neurotransmitter release by |
|
Definition
|
|
Term
| E acts on alpha1AR to cause vasoconstriction by |
|
Definition
| Activating phospholipase C |
|
|
Term
| Calcitriol is described as a |
|
Definition
|
|
Term
| The vitamin D receptor is a |
|
Definition
|
|
Term
| There are no know clinical symptoms associated with this hormone |
|
Definition
|
|
Term
| Which hormone decreases serum K levels |
|
Definition
|
|
Term
| Which of the following will not increase insulin release |
|
Definition
|
|
Term
| The kinetic properties of glut2 and the glucokinase in beta cells produces a rate of glycolysis and ATP production that is |
|
Definition
| Directly proportional to the blood glucose concentration |
|
|
Term
| Glucose transport molecule found on the liver, kidney, and beta cells of the pancreas |
|
Definition
|
|
Term
| Glucose transport molecule found on muscle and fat cells |
|
Definition
|
|
Term
| Glucose transport molecule found on most cells |
|
Definition
|
|
Term
| These GI hormones: CCK, GIP, gastrin stimulate the release of what |
|
Definition
|
|
Term
| SNS acting on beta2 receptors stimulate the release of what |
|
Definition
|
|
Term
| Hypoglycemia, stress, exercise, and AA stimulate the release of what |
|
Definition
|
|
Term
| Carbs, ffa, and ketoacids inhibit the release of what |
|
Definition
|
|
Term
| GI hormones GLP1 and secretin inhibit the release of what |
|
Definition
|
|
Term
| Which of the following is stimulates insulin release |
|
Definition
|
|
Term
| CRH, GnRH, GHRH, GHIH, TRH |
|
Definition
|
|
Term
| Simulated by GHRH, this hormone Increases glucose, fatty breakdown (fatty acid, glycerol) |
|
Definition
|
|
Term
| Main thing that stimulates GHRH |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| Increases transcription of mitochondria, Na/K, betaAR, O2 consumption |
|
Definition
|
|
Term
| Hypothyroidism effect on HR |
|
Definition
|
|
Term
| Hyperthyroidism effect on HR |
|
Definition
| Tachycardia and possibly thin |
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| (-) feedback on CRH, ACTH |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| Increases: glucose, glycogen synthase, betaAR, CV output. Maintains muscle contraction but can decrease muscle protein and is an immunosuppressent |
|
Definition
|
|
Term
| Causes low K, high Na and is inhibited by ANP |
|
Definition
|
|
Term
| Glucagon, GH, E, Cortisol are stimulated by |
|
Definition
|
|
Term
| Levels are highest right before waking up and lowest before bed |
|
Definition
|
|
Term
|
Definition
|
|
Term
| May lead to metabolic alkalosis and hypokalemia |
|
Definition
|
|
Term
| Directly stimulates FSH, LH |
|
Definition
|
|
Term
| Directly stimulates leydig cells |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Stimulates growth of the male genitalia |
|
Definition
|
|
Term
| Increases muscle mass, bone mass, Blood cells, growth of reproductive tissues, important in spermatogenesis |
|
Definition
|
|
Term
| (-) feedback on GnRH, LH, FSH |
|
Definition
|
|
Term
| Converted into estradiol or DHT |
|
Definition
|
|
Term
| Important for male external genitalia, male pattern baldness, bone/cartilage growth |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Sertoli cells release this hormone that inhibits formation of internal female genitalia |
|
Definition
|
|
Term
| Male Spermatogonia undergo mitosis at around this age |
|
Definition
|
|
Term
| Spermatogonia undergo mitosis forming |
|
Definition
|
|
Term
| Phase that primary spermatocytes undergo includes DNA replication, tetrad crossover, separation of chromosomes |
|
Definition
|
|
Term
| Primary spermatocytes divide forming |
|
Definition
|
|
Term
| Phase that secondary spermatocytes undergo with no DNA replication but chromosome separation forming haploid spermatids |
|
Definition
|
|
Term
| An erection is caused by PNS secreting ACh and NO causing release of what second messenger |
|
Definition
|
|
Term
| Ejaculation involves the SNS and |
|
Definition
|
|
Term
| In females, all oogonia form Primary oocytes by this age |
|
Definition
|
|
Term
| Primary oocytes enter Meiosis I forming secondary oocytes at this age |
|
Definition
|
|
Term
| Secondary oocytes enter meiosis II only when |
|
Definition
|
|
Term
| High CRH, High ACTH, Low cortisol, Low Aldosteron, Low DHEA |
|
Definition
|
|
Term
|
Definition
|
|
Term
| High CRH, High ACTH, Low cortisol, Low Aldosteron, High DHEA |
|
Definition
| Congenital adrenal hyperplasia |
|
|
Term
| Pregnant women with low cortisol levels can lead to this disease |
|
Definition
| Congenital adrenal hyperplasia |
|
|
Term
| Disease causing high aldosterone |
|
Definition
|
|
Term
| Low CRH, High ACTH, High cortisol, Normal Aldosteron |
|
Definition
|
|
Term
| Low CRH, Low ACTH, Low cortisol,Normal Aldosteron |
|
Definition
| Cushings syndrome (Iatrogenic) |
|
|
Term
| High TRH, High TSH, Low total T3/4, Low free T3/4 |
|
Definition
| Hypothyroidism (Hashimotos) |
|
|
Term
| High TRH, High TSH, Low total T3/4, Low free T3/4 |
|
Definition
|
|
Term
| Low TRH, Low TSH, High total T3/4, High free T3/4 |
|
Definition
|
|
Term
| How do you test for graves |
|
Definition
|
|
Term
| Low TRH, High TSH, High total T3/4, High free T3/4 |
|
Definition
|
|
Term
| Low TRH, Low TSH, High total T3/4, High free T3/4 |
|
Definition
|
|
Term
| Low TRH, Low TSH, High total T3/4, High free T3/4 |
|
Definition
|
|
Term
| How do you test for a thyroid tumor |
|
Definition
|
|
Term
| N TRH, N TSH, High total T3/4, N free T3/4 |
|
Definition
|
|
Term
| MWF, 48, palpitations, sweating, trumors, anxiety, constant hunger and weight gain, confusion and increased anxiety without food. What are these symptoms of |
|
Definition
|
|
Term
| High glucose, high insulin, high c peptide, low glucagon. What is the correct diagnosis |
|
Definition
| Hypoglycemia from an insulin secreting beta cell tumor |
|
|
Term
| How do peptides like leptin and insulin cross the blood brain barrier |
|
Definition
| Receptor mediated endocytosis |
|
|
Term
| High fat> +Leptin> stimulates ____ |
|
Definition
|
|
Term
| High fat> +Leptin> inhibit ____ |
|
Definition
|
|
Term
| What is a receptor anatagonist |
|
Definition
| Blocks the effect of an endogenous ligand |
|
|
Term
| Which hypothalamic neurotransmitter is stimulated by leptin |
|
Definition
|
|
Term
| Which hypothalamic neurotransmitter acts an an endogenous agonist of the melanocortin receptor |
|
Definition
|
|
Term
| Which hypothalamic neurotransmitter acts an an endogenous antaagonist of the melanocortin receptor |
|
Definition
|
|
Term
| Which of the following acts as an endocannabinoid |
|
Definition
|
|
Term
| GH deficiency leads to this |
|
Definition
| Dwarfism with normal proportions |
|
|
Term
| GH secreting tumor before puberty leads to this |
|
Definition
|
|
Term
| GH secreting tumor after puberty leads to this |
|
Definition
|
|
Term
| GH secreting tumor leads to |
|
Definition
| Enlarged bones and organs |
|
|
Term
| GnRH is released from what type of cells |
|
Definition
|
|
Term
| Directly stimulates theca cells |
|
Definition
|
|
Term
| Stimulates the corpus luteum |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Hormone stimulating theca cells to release hormones for ovulation |
|
Definition
|
|
Term
| Stimulates granulosa cells |
|
Definition
|
|
Term
| Converts androgens to estrogen |
|
Definition
|
|
Term
| Stimulates ganulosa cells to release aromatase |
|
Definition
|
|
Term
| Stimulates the release of estrogen |
|
Definition
|
|
Term
| First stage in the menstrual cycle is known as |
|
Definition
|
|
Term
| Following the beginning of menses estrogen stimulates this next phase |
|
Definition
|
|
Term
| Phase taking place in the uterus from estrogen produced in a follicle of the ovary |
|
Definition
| Follicular phase of proliferation |
|
|
Term
| During the proliferation phase this hormone negatively feedbacks on LH, FSH |
|
Definition
|
|
Term
| Estrogen produced from the dominate follicle produces a temporary dominate feedback on GnRH causing the next phase called |
|
Definition
|
|
Term
| The ____ ____ causes the corpus luteum to stay behind |
|
Definition
|
|
Term
| LH surge causes the corpus luteum to produce high levels of progesterone in this phase |
|
Definition
|
|
Term
| During the secretory phase ____ stimulates maintenance of the vaginal mucosa, cervical secretion, breast development, all favoring intercourse |
|
Definition
|
|
Term
| If you do have pregnancy, outer synctiotrophoblasts secrete this hormone |
|
Definition
|
|
Term
| Placenta is made up of these cells |
|
Definition
| Syncytiotrophoblasts and cytotrophoblasts |
|
|
Term
| hGC binds receptors on the corpus luteum to produce what |
|
Definition
| Estrogen and progesterone |
|
|
Term
| hCG causes the secretory phase to change to what phase |
|
Definition
|
|
Term
| Corpus luteum is important in which trimester |
|
Definition
|
|
Term
| The placenta takes over from the corpus luteum in which trimester |
|
Definition
|
|
Term
| Placental hormone with GH like effects stimulating breast development and glucose formation if mother doesn't produce enough |
|
Definition
|
|
Term
| If mother doesnt repsond to chorionosomatotrophin (CS) this can lead to |
|
Definition
|
|
Term
| Stimulates prolactin release from mothers pituitary |
|
Definition
|
|
Term
| Stimulate breast development |
|
Definition
| Estrogen and progesterone |
|
|
Term
| Closer to delivery this hormone downregulates its receptors |
|
Definition
|
|
Term
| Closer to delivery there is upregulation of this hormones receptors |
|
Definition
|
|
Term
| Close to delivery this hormone stimulates smooth muscle contraction of the myometrium, lactiferous breast duct myoepithelial cells |
|
Definition
|
|
Term
| During labor, stretch on cervix causes positive feedback on this hormone until fetus and placenta are delivered |
|
Definition
|
|
Term
| During breast feeding suckling causes feedback on the hypothalamus to secrete ___ and inhibit ____ |
|
Definition
|
|
Term
| Prolactin inhibiting hormone |
|
Definition
|
|
Term
| Prolactin inhibiting hormone |
|
Definition
|
|
Term
| Which of the following is not a direct action of GH |
|
Definition
|
|
Term
| Which of the following is a direct action of GH |
|
Definition
| Increase, blood glucose, IGF1 synthesis, lipolysis |
|
|
Term
|
Definition
|
|
Term
| Released from somatotrophs |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Released from tyroptrophs |
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
|
Definition
|
|
Term
| Released from thyroid gland |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Released from corticotrophs |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Released from the adrenal gland |
|
Definition
|
|
Term
|
Definition
|
|
Term
| Released from gonadotrophs |
|
Definition
|
|
Term
| Regulates metabolic rate of virtually every tissue in the body |
|
Definition
|
|
Term
| Increases O2 consumption, heat production, Na/K ATPase, number of mitochondria |
|
Definition
|
|
Term
| T3/4 effect on beta1 adrenergic receptors |
|
Definition
|
|
Term
| Where is vasopressin synthesized |
|
Definition
|
|
Term
| Where is vasopressin released |
|
Definition
|
|
Term
| Graves disease with elevated blood levels of thyroid hormone and thyroid stimulating Ig. What would you except of her TSH levels |
|
Definition
|
|
Term
| Elevated thyroid hormone blood levels with low levels of TSH. Which condition |
|
Definition
|
|
Term
| low thyroid hormone blood levels with elevated TSH. Which condition |
|
Definition
|
|
Term
| Low thyroid hormone blood levels with very low levels of TSH. Which condition and, Following surgery to remove the ___ ___ Which of the following replacement hormones should he receive |
|
Definition
| Pituitary tumor, thyroid hormone |
|
|
Term
| Where is cortisol synthesized |
|
Definition
|
|
Term
| Where is ACTH synthesized |
|
Definition
| Corticotrophs of the anterior pituitary |
|
|
Term
|
Definition
| Neurons in the hypothalamus |
|
|
Term
| Which of the following stimulates CRH release |
|
Definition
|
|
Term
| Which of the following is not an effect produced by cortisol |
|
Definition
|
|
Term
| Which directly binds a G protein receptor |
|
Definition
|
|
Term
| Which of the following is stimulates more by E than NE |
|
Definition
| Beta2 smooth muscle vasocontriction |
|
|
Term
| Which signal transduction system does ACh directly activate to produce its effects at nicotinic cholinergic receptors |
|
Definition
|
|
Term
| Which signal transduction system does ACh activate to produce its effects at muscarinic cholinergic receptors |
|
Definition
|
|
Term
| Alpha1 ADR activate PLC to increase ____ levels to produce _____ |
|
Definition
| Ca, smooth muscle contraction |
|
|
Term
| Alpha2 ADR predominately inhibits ____ decreasing neurotransmitters |
|
Definition
|
|
Term
| All the beta ADR predominately increase |
|
Definition
|
|
Term
| Which of the following directly prevents formation of the uterus |
|
Definition
|
|
Term
| Which of the following stimulates formation of the testes |
|
Definition
|
|
Term
| Which of the following stimulates the formation of the seminal vesicles |
|
Definition
|
|
Term
| Which of the following stimulates the formation of the male penis |
|
Definition
|
|
Term
| Which of the following synthesizes testosterone |
|
Definition
|
|
Term
| Which of the following stimulates testosterone synthesis |
|
Definition
|
|
Term
| Which of the following stimulates spermatogenesis |
|
Definition
|
|
Term
| Which of the following undergoes meiosis II |
|
Definition
|
|
Term
| Tetrad formation and crossing over occur when which of the following divide |
|
Definition
|
|
Term
| Which of the following directly stimulates spermatogenesis |
|
Definition
|
|
Term
| Intermittent release of GnRH effect on LH, FSH |
|
Definition
|
|
Term
| Which of the following synthesis androgens |
|
Definition
|
|
Term
| Which of the following synthesis estrogens |
|
Definition
|
|
Term
| When do oogonia stop mitosis |
|
Definition
|
|
Term
| When do spermatogonia stop mitosis |
|
Definition
|
|
Term
| When do primary oocytes form |
|
Definition
|
|
Term
| When do primary spermatocytes form |
|
Definition
| 12 years of age until rigor mortis |
|
|
Term
| When do secondary spermatocytes form |
|
Definition
| 12 years of age until rigor mortis |
|
|
Term
| When do secondary oocytes form |
|
Definition
| 12-50 years of age or at ovulation |
|
|
Term
| When do secondary oocytes complete meiosis II |
|
Definition
|
|
Term
| What type of receptor do estrogens act on |
|
Definition
|
|
Term
| Which of the following directly stimulates proliferation of the endometrium |
|
Definition
|
|
Term
| Which of the following directly stimulates the secretory actions of the endometrium |
|
Definition
|
|
Term
| Which of the following stimulates ovulation |
|
Definition
|
|
Term
| Which of the following stimulates the granulosa cells to divide and secrete estrogen |
|
Definition
|
|
Term
| Which of the following stimulates the theca cells to divide and secrete androgens |
|
Definition
|
|
Term
| Which of the following directly stimulates the formation of the corpus luteum and the secretion of progesterone |
|
Definition
|
|
Term
| Posterior pituitary hormones |
|
Definition
|
|
